Art Forgeries Essay Example for Free

Art Forgeries Essay When one enters into an art museum, one would expect all of the pieces of art to be that of the original. However, when an art lover does not know the difference between an original from a forgery, then they may have been fooled by both the museum and by the forger. No one can really look at a painting and distinctly know whether it is a forged piece of work or an original piece of work. Art forgeries may seem like an artist copying a well-known artists work, but it depends on how one looks at a particular piece of art. There are multiple reasons as to why art forgeries can be seen as something positive in the artistic world. Crispin Sartwell discusses about Jerrold Levinson’s definition of art from Levinson’s article, â€Å"Refining Art Historically,† in the Journal of Aesthetics Art Criticism of 1990. To Levinson, art is something that is made to be intended to be â€Å"regarded† as a work of art (Sartwell). Luise Morton and Thomas Foster discuss Nelson Goodman’s definition of art from Languages of Art in Journal of Aesthetics Art Criticism of 1991. Goodman says that it is about how one looks at an original and a forged art depends on the way we look at it (Morton and Foster). Both Levinson and Goodman make good points because they are both saying that all depends on the person’s perspective. Not everyone sees the same piece of art the same way another person does, so given a choice between an original or a forged piece of art, some may be able to tell the difference and some may not. The idea of having different perspectives on what is real art or what is not depends solely on an individual. Levinson and Goodman both see art by how the person intends it to be. According to Jonathon Keats who writes in The Daily Beast, art forgery helps take us out of our comfort zone, while the real art keeps us within our comfort zone. Keats writes that forgers credit their work to the original artist. In doing so, the artist’s work is more accessible to more people and that the artist who forged an original should be appreciated (Keats). Blake Gopnik writing in the New York Times says that the forgers can make recreate art with their hands; however, great art depends on the idea of the artist. The idea of the forger comes from the original artists, like Pollock and Rothko, setting up procedures and ideas for making art (Gopnik). The forger is able to recreate a work of art because of the way a particular artist wanted their art to be seen. On the other hand, Ross Bowden writing in the Journal of Aesthetics Art Criticism of 1999 about Alfred Lessing’s essay titled, â€Å"What Is Wrong with a Forgery? † In Lessing’s essay, he disapproves of art forgery when talking culturally. Lessing believes that forgeries do not have that artistic integrity and lacks creativity. He continues to say that one can recreate an amazing artwork, but it will lack the imagination it takes to create the original piece of work (Bowden). Forgeries in the opinion of Lessing lack imagination and creativity, however, Gopnik and Keats see that an artist has the imagination and creativity to recreate a famous piece of work. If one lacks that imagination and creativity then they would not be able to get away with forgery. These forgery artists are capable of pulling off century old paintings and able to sell them to museums as originals. That takes imagination and creativity. W. E. Kennick brings up in the Journal of Aesthetics Art Criticism of 1985 that every copy of an original piece of work is a forgery. Artists make their work in the style of others, but still make it their own. One is not actually forging a real piece of art, much less than copying or imitating that person’s style (Kennick). Gopnik also says that Andy Warhol’s works were sometimes made by him or sometimes made by his assistant. Warhol even attributed some of his work to other artists. An artist by the name of Marcel Duchamp made art out of bicycle wheels, urinals, already made sculptures, and other reusable items. Duchamp encouraged others to do the same and copy his style (Gopnik). Every artist can imitate or copy someone else’s work, although that artist who made the original work may no longer be alive, their work is still living on. Art forgeries can be looked at as some sort of crime because someone is recreating masterpieces and selling them to museums. However, if one stops to think about the fact that art forgeries are actually artists bringing masterpieces back, one would not think it was a crime. These artists are creative enough to be able to recreate an artwork and give art lovers the feeling of having a masterpiece in their home or be able to look at it in a museum. Art forgeries are a lucrative past time for those who love art and want to be able to see â€Å"their art† in a museum. It is a win-win situation for both the artist and the art lovers. Works Cited Bowden, Ross. What is wrong with an art forgery? : An anthropological perspective. Journal of Aesthetics Art Criticism (1999): 333-343. Gopnik, Blake. In Praise of Art Forgeries . The New York Times 2 Novemeber 2013. Keats, Jonathon. Why Forgeries Are Great Art. The Daily Beast Kennick, W. E. Art and Inauthenticity. Journal of Aesthetics Art Criticism (1985): 1-12. Morton, Luise H. and Thomas R. Foster. Goodman, Forgery, and the Aesthetic. Journal of Aesthetics Art Criticism (1991): 155-159. Sartwell, Crispin. A Counter-Example to Levinsons Historical Theory of Art. Journal of Aesthetics Art Criticism (1990): 157-158.

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Jason Obertin-Andersen Impact of theft on consumers and businesses I think that shoplifting is a growing problem in today's society. I believe that the temptation of not paying for something is very big factor in why people steal. Shoplifters perceive shoplifting as a form of self nourishment or as a way to relieve fear or pain in their life. In truth, shoplifting is self-destructive not self-nourishing, but shoplifters often can’t see the paradox (slp). A person's addiction to shoplifting can develop quickly when the excitement generated from "getting away with it" produces a chemical reaction adrenaline resulting in what shoplifters describe as an incredible "rush" or "high" feeling, which many shoplifters will tell you is the "true reward," rather than the merchandise itself (slp). The perpetrator of shoplifting thinks he his getting a product for free and doesn't realize it's impact on his future or the community. Shoplifting has a wide range of affects on more than just the perpetrator and his future. It negatively affects the merchan t, consumers, prices, law enforcement and the economy. How does shoplifting affect consumers? According to the National Association for Shoplifting Prevention, â€Å"Shoplifting has become one of the most prevalent crimes in the U.S., averaging about 550,000 incidents per day resulting in more than $13 billion worth of goods being stolen from retailers each year. That is more than 35 million dollars in losses per day. Current estimates are as high as 1 in 11 Americans shoplift in our nation today. Even with all the advances in security measures, shoplifters are only caught once in 49 times they steal and when caught, turned over to the police only 50% of the time. What most people don’t real... ...ation, said that the overriding concern is not endangering employees or customers around them when trying to confront shoplifting. The policy at most major retailers is to simply let the thief go if he or she does not comply with more passive anti-theft maneuvers, such as friendly inquiries as to whether the suspected shoplifter can be helped while in the store. This means that by and large, low-level criminals are simply left undisturbed, in order to avoid escalating costs and liability. Police officers and the court system are concerned with all sorts of different crimes, and the focus is mainly on the crimes that most severely impact their communities. While shoplifting may seem a minor offense in comparison to other crimes, it is still important to realize the impact that shoplifting has on the law enforcement agencies, the community and even on the shoplifter.

Manifestations of Dementia

Manifestations of Dementia Melinda Godfrey GNUR543 St. John Fisher College Mrs. Yowell is a 90-year-old woman who is a resident of a long-term care facility. She was alert and mentally quite capable until about a year ago when she began to manifest signs and symptoms of dementia. A review of her medical records failed to document a thorough analysis of her dementia, but a diagnosis of â€Å"probable Alzheimer disease† was recorded. What are the common manifestations of dementia? The definition of Dementia is â€Å"a general term for loss of memory and other mental abilities severe enough to interfere with daily life.It is caused by physical changes in the brain. † (Common Types of Dementia, 2012). The first manifestations of Dementia usually are: * Loss of memory – generally the patient doesn’t notice the loss of memory but a loved one will. This is generally the entree into the physician’s office leading to a diagnosis. * Trouble focusing and follo wing conversations – the patient is unable to handle more than one task at a time and will not be able to perform a task and listen to or follow a conversation easily. * Impaired judgment and reasoning – patients become confused and are unable to cope as well when unexpected events come up.Other symptoms could include: mood changes, personality and behavior changes. As noted, dementia is not a disease itself but a cluster of cognitive changes. The onset symptom of dementia can vary depending on the disease or syndrome that is associated (Common Types of Dementia, 2012). The major dementia diseases or syndromes are: 1. Alzheimer Disease (AD) – AD accounts for 60 – 80% of the cases of dementia (Shadlen & Larson, 2012). 2. Dementia with Lewy Bodies (DLB) – DLB starts with progressive cognitive decline and usually the patient also has hallucinations, periods of lucidity and some rigidity.DLB has abnormal accumulations of protein structures in the patie nt’s brain (Dementia With Lewy Bodies Information Page, 2011). 3. Frontotemporal Dementia (FT) – FT is associated with the shrinking of the frontal and temporal anterior lobes of the brain (Frontotemporal Dementia Information Page, 2011). Generally the patient will have trouble with speech and behavior. 4. Vascular Dementia – This form of dementia results from many strokes. When a patient has a stroke, the blood flow is interrupted to the brain and the result is brain tissue damage (Types of Dementia, 2011).These patients normally lose their cognitive ability before their memory. 5. Parkinson Disease with Dementia – Approximately 40% of patients with Parkinson’s disease will have dementia (Parkinson’s disease, 2011). The nerve cells in the brain that make dopamine are slowly destroyed leaving no way for the brain to send messages. What other potentially treatable factors might have led to Mrs. Yowell’s deteriorating mental function? T here are many reasons that Mrs. Yowell may have impaired mental functioning (see Table 1). The first and easiest reason to rule out would be a urinary tract infection (UTI).A urine sample can show if there are white blood cells (indicating infection) present in the urine. The increased levels of bacteria in an elderly person’s body can cause toxicity which leads to the altered mental status (Midthun, 2004). Most noted changes are confusion, agitation and lethargy. Table 1| | (Shadlen & Larson, 2012)| Secondly, Mrs. Yowell could have vitamin deficiencies. For example, if a patient has low levels of thiamine they can develop Wernicke’s encephalopathy. This can cause damage in the brain to the thalamus and the hypothalamus.These patients will exhibit signs of confusion, loss of memory and hallucinations (Dugdale, 2010). If left untreated it can cause permanent damage but, it is usually corrected by injections of thiamine. Another cause of confusion/delirium in elderly pat ients is an adverse drug reaction or interactions with another drug. Mrs. Yowell could have been given a new medication that is directly affecting her mental status. Nearly 70% of residents in long term facilities take nine or more medications (Gillick, 2012). Elderly patients have slower clearance rates of drugs through their liver and kidney thus leading to drug accumulation (Gillick, 2012).There should be a regular review of medications in order to anticipate and keep on top of any potential changes. Discuss in detail the organic brain/neuronal alterations that are typical of Alzheimer disease? The changes that occur in the brain with Alzheimer’s disease (AD) are threefold. The first is amyloid plaque, this is abnormal clumps of protein found outside of the nerve cells. The protein pieces are in the fatty membrane that is around the brains nerve cells. The amyloid is a protein that is snipped from a larger protein (amyloid precursor protein) during metabolism (Copstead, 20 10).These clumps form plaque and can block cell-to-cell signaling (Common Types of Dementia, 2012). In a healthy brain the fragments are broken down and eliminated before they turn into plaque. Without the ability to signal the brain may also activate the inflammatory response. At this point it is still not known whether the plaque causes or is created by AD. Secondly, inside the nerve cells there is a protein called tau. This protein is normally helps maintain the cell structure allowing proper nutrition and signaling. The tau protein in AD is changed into twisted strands, called tangles (Common Types of Dementia, 2012).It is said that the changes are brought on by the phosphorylating enzymes which are activated by inflammatory changes, lipid abnormalities and aging (Copstead, 2010). Thirdly, the brain is losing connectivity between nerve cells and cells are dying which results in permanent brain damage. Other changes in the brain include shrinking of the hippocampus and cortex. AD also shows an increase in ventricles. The progression of the damage in the AD brain is predictable. According to the Alzheimer’s research center, the plaques and tangles spread through the cortex in a predictable fashion (Common Types of Dementia, 2012).Summary Scientists believe that the cause of AD is multi-factorial. IT could be a combination of genetics, lifestyle and environmental factors. Specifically, it is believed that a person with diabetes, depression or heart disease is more likely to develop AD (Copstead, 2010). There is a link to a gene, APOe4 that is thought to be associated with late onset AD. However, studies have shown it is not present in some cases of AD and is present in cases with no AD (Common Types of Dementia, 2012). At this point the definitive diagnosis of AD can only be obtained after death (Common Types of Dementia, 012). This means that clinicians must ask the right questions to diagnose this disease. AD is still quite unknown as to the order of the changes that occur in the brain. As more research is conducted we will be able to identify the manifestations of the disease earlier and slow the progression of AD. References Common Types of Dementia. (2012). Retrieved March 14, 2012, from Alzheimer's Association: http://www. alz. org Copstead, L. -E. C. (2010). Pathophysiology. St. Louis: Saunders. Dementia With Lewy Bodies Information Page. (2011, December 28).Retrieved March 8, 2012, from National Institute of Neurological Disorders and Stroke: http://www. ninds. nih. gov Dugdale, I. M. (2010, February 6). Wernicke-Korsakoff Syndrome. Retrieved March 10, 2012, from Medline Plus: http://www. nlm. nih. gov/medlineplus Frontotemporal Dementia Information Page. (2011, December 28). Retrieved March 8, 2012, from National Institute of Neurological Disorders and Stroke: http://www. ninds. nih. gov Gillick, M. M. (2012, February 22). Medical Care of the Nursing Home Patient in the United States. Retrieved March 10, 2012, from UpToD ate: http://www. uptodate. com Lexicomp. 2012). Retrieved February 5, 2012, from Lexicomp: https://online-lexi. com Midthun, M. R. (2004). Criteria for Urinary Tract Infections. Retrieved March 4, 2012, from Medscape: http://www. medscape. com Parkinson’s disease. (2011, September 26). Retrieved March 5, 2012, from PubMed Health: http://www. ncbi. nlm. nih. gov Shadlen, M. -F. M. , ; Larson, E. B. (2012, February 10). Evaluation of Cognitive Impairment and Dementia. Retrieved March 5, 2012, from UpToDate: http://www. uptodate. com Types of Dementia. (2011, June 23). Retrieved March 14, 2012, from Cleveland Clinic: http://my. clevelandclinic. org

Louisa May Alcotts Family Tree and Genealogy

Louisa May Alcott, best known as the author of Little Women, never married and has no descendants. Her rich ancestry, however, stretches back to early America and Europe and includes many well-known people, including her father, famous transcendentalist Bronson Alcott. Many people can claim a relation to Louisa May Alcott through her siblings, cousins and other relatives. Born on November 29, 1832 in Germantown, Pennsylvania (now a part of Philadelphia), Louisa May Alcott was the second of four girls born to Bronson Alcott and his wife, Abigail May. The March family everyone came to love in her books is based on her own family, with Louisa as her alter-ego Jo and her sisters as the other three little women. Louisa May Alcott died just two days after her father, on March 4, 1888 from long-term side-effects of mercury poisoning. She initially acquired this disorder from the drug calomel (which is laden with mercury) that doctors used to treat the typhoid fever she contracted while volunteering as a nurse during the Civil War. Louisa May Alcott is buried on Authors Ridge in Concords Sleepy Hollow Cemetery, with her family. Nearby, are the graves of Ralph Waldo Emerson, Nathaniel Hawthorne, and Henry David Thoreau. First Generation Reading the Ahnentafel genealogical numbering system is not difficult once you understand how this family tree is arranged. 1. Louisa May ALCOTT was born on 29 Nov 1832 in Germantown, Philadelphia, Pa. and died on 6 Mar 1888 in Boston, Suffolk Co., Ma. Second Generation (Parents) 2. Amos Bronson ALCOTT was born on 29 Nov 1799 in Wolcott, New Haven, Ct. and died on 4 Mar 1888. He married Abigail MAY on 23 May 1830. 3. Abigail MAY was born on 8 Oct 1800 in Boston, Suffolk Co., Ma. and died in 1877. Amos Bronson ALCOTT and Abigail MAY had the following children: i. Anna Bronson ALCOTT was born on 16 Mar 1831 in Germantown, Philadelphia, Pa.1 ii. Louisa May ALCOTTiii. Elizabeth Sewall ALCOTT was born on 24 Jun 1835 in Boston, Suffolk Co., Ma. and died on 14 Mar 1858.iv. May ALCOTT was born on 26 Jul 1840 in Concord, Middlesex Co., Ma. Third Generation (Grandparents) 4. Joseph Chatfield ALCOTT was born on 7 May 1771 in Wolcott, New Haven, Ct. and died on 3 Apr 1829. He married Anna BRONSON on 13 Oct 1796 in Wolcott, New Haven, Ct. 5. Anna BRONSON was born on 20 Jan 1773 in Jerico, New London, Ct. and died on 15 Aug 1863 in West Edmeston, Ostego Co., New York. Joseph Chatfield ALCOTT and Anna BRONSON had the following children: i. Betsey ALCOTT was born on 4 Apr 1798 in Wolcott, New Haven, Ct. and died on 5 Nov 1798.2 ii. Amos Bronson ALCOTTiii. Chatfield ALCOTT was born on 23 Oct 1801.iv. Pamelia ALCOTT was born on 4 Feb 1805 in Wolcott, New Haven, Ct. and died on 11 Feb 1849.v. Betsey ALCOTT was born on 14 Feb 1808 in Wolcott, New Haven, Ct.vi. Phebe ALCOTT was born on 18 Feb 1810 in Wolcott, New Haven, Ct. and died on 28 Jul 1844.vii. George ALCOTT was born on 26 Mar 1812 in Wolcott, New Haven, Ct. and died on 12 Jul 1812.viii. Junius ALCOTT was born on 6 Jul 1818 and died on 16 Apr 1852.ix. Ambrose ALCOTT was born on 10 Sep 1820 in Wolcott, New Haven, Ct. 6. Joseph MAY was born on 25 Mar 1760 in Boston, Suffolk Co., Mass. and died on 27 Feb 1841 in Boston, Suffolk Co., Mass. He married Dorothy SEWELL on 28 Dec 1784 in Boston, Suffolk Co., Mass. 7. Dorothy SEWELL was born on 23 Dec 1758 in Boston, Suffolk Co., Mass. and died on 31 Oct 1825 in Boston, Suffolk Co., Mass. Joseph MAY and Dorothy SEWELL had the following children: i. Charles MAY was born on 2 Nov 1785 in Roxbury, Norfolk Co., Mass. and died on 21 Mar 1856 in Roxbury, Norfolk Co., Mass.ii. Catharine MAY was born on 30 Dec 1786 in Boston, Suffolk Co., Mass. and died in 1814 in Boston, Suffolk Co., Mass.iii. Louisa MAY was born on 31 Dec 1792 in Roxbury, Norfolk Co., Mass. and died on 14 Nov 1828 in Roxbury, Norfolk Co., Mass.iv. Edward MAY was born on 26 Aug 1795 in Roxbury, Norfolk Co., Mass. and died on 29 Apr 1802 in Roxbury, Norfolk Co., Mass.v. Samuel Joseph MAY was born on 12 Sep 1797 in Roxbury, Norfolk Co., Mass. and died on 1 Jul 1871 in Roxbury, Norfolk Co., Mass.vi. Elizabeth Sewall MAY was born on 5 Dec 1798 in Boston, Suffolk Co., Mass. and died on 5 Mar 1822 in Portland, Cumberland Co., Maine.3 vii. Abigail MAYviii. Louisa C. Greenwood MAY was born on 2 Dec 1810 in Roxbury, Norfolk Co., Mass. and died on 23 Sep 1891 in Roxbury, Norfolk Co., Mass. Fourth Generation (Great Grandparents) 8. Captain John  ALCOX  was born on 28 Dec 1731 in Wolcott, New Haven, Conn. and died on 27 Sep 1808 in Wolcott, New Haven, Conn. He married Mary CHATFIELD on 28 Aug 1755 in Connecticut. 9. Mary CHATFIELD was born on 11 Oct 1736 in Derby, New Haven, Conn. and died on 28 Feb 1807 in Wolcott, New Haven, Conn. She was christened 7  Noc  1736 in First Congregational Church of Derby. Captain John ALCOX and Mary CHATFIELD had the following children: i. Lydia ALCOTT was born on 8 Dec 1756 in Wolcott, New Haven, Conn. and died on 23 Sep 1831.ii. Solomon ALCOTT was born on 8 May 1759 in Wolcott, New Haven, Conn. and died on 21 May 1818 in Wolcott, New Haven, Conn.iii. Samuel ALCOTT was born on 29 Nov 1761 in Wolcott, New Haven, Conn. and died on 9 Jun 1819.iv. John Blakeslee ALCOTT was born on 24 Jun 1764 in Wolcott, New Haven, Conn. and died on 17 Sep 1837.v. Mary ALCOTT was born on 8 Sep 1766 in Wolcott, New Haven, Conn. and died on 18 Feb 1770.vi. Isaac ALCOTT was born on 12 Apr 1769 in Wolcott, New Haven, Conn. and died on 12 Sep 1809.4  vii.  Joseph Chatfield ALCOTTviii. Mark ALCOTT was born on 11 May 1773 in Wolcott, New Haven, Conn. and died on 21 Nov 1846.ix. Thomas ALCOTT was born on 16 Oct 1775 and died on 27 Apr 1778. 10. Amos BRONSON was born on 3 Feb 1729/30 in Waterbury, New Haven, Conn. and died on 2 Sep 1819 in Waterbury, New Haven, Conn. He married Anna BLAKESLEY on 3 Jun 1751 in Waterbury, New Haven, Conn. 11. Anna BLAKESLEY was born on 6 Oct 1733 in New Haven, New Haven, Conn. and died on 3 Dec 1800 in Plymouth, Litchfield, Conn. Amos BRONSON and Anna BLAKESLEY had the following children: i. Noah Miles BRONSON was born on 15 Jul 1767 in Waterbury, New Haven, Conn. and died on 8 Sep 1859 in Weymouth, Medina Co., Ohio.5  ii.  Anna BRONSON 12. Samuel MAY was born. He married Abigail WILLIAMS. 13. Abigail WILLIAMS was born. Samuel MAY and Abigail WILLIAMS had the following children: 6  i.  Joseph MAY 14. Samuel SEWELL was born on 2 May 1715 in Boston, Suffolk Co., Mass. and died on 19 Jan 1771 in Holliston, Middlesex Co., Mass. He married Elizabeth QUINCY on 18 May 1749 in Boston, Suffolk Co., Mass. 15. Elizabeth QUINCY was born on 15 Oct 1729 in Quincy, Norfolk Co., Mass. and died on 15 Feb 1770. Samuel SEWELL and Elizabeth QUINCY had the following children: i. Elizabeth SEWELL was born on 12 Mar 1750 and died in 1789.ii. Samuel SEWELL was born on 11 Dec 1757 in Boston, Suffolk Co., Mass. and died on 7 Jun 1814 in Wiscasset, Lincoln Co., Maine.7  iii.  Dorothy SEWELL Fifth Generation (Great, Great Grandparents) 16. John ALCOCK was born on 14 Jan 1705 in New Haven, New Haven, Conn. and died on 6 Jan 1777 in Wolcott, New Haven, Conn. He married Deborah BLAKESLEE on 14 Jan 1730 in North Haven, New Haven, Conn. 17. Deborah BLAKESLEE was born on 15 Mar 1713 in New Haven, New Haven, Conn. and died on 7 Jan 1789 in Wolcott, New Haven, Conn. John ALCOCK and Deborah BLAKESLEE had the following children: i. Lydia ALCOTT was born on 24 Nov 1730 in North Haven, New Haven, Conn. and died on 15 Nov 1796 in North Haven, New Haven, Conn.8  ii.  Captain John ALCOXiii. James ALCOTT was born on 1 Jun 1734 in Waterbury, New Haven, Conn. and died on 9 Aug 1806.iv. Jesse ALCOTT was born on 23 Mar 1736 in Waterbury, New Haven, Conn. and died on 29 Oct 1809.v. Daniel ALCOTT was born on 25 Mar 1738 in Waterbury, New Haven, Conn. and died on 24 May 1805.vi. David ALCOTT was born on 12 Jan 1740 in Waterbury, New Haven, Conn. and died on 29 Jan 1821.vii. Deborah ALCOTT was born in 1742 in Waterbury, New Haven, Conn. and died on 18 Jun 1831.viii. Mary ALCOTT was born in 1744 in New Haven, New Haven, Conn. and died on 6 Mar 1825.ix. Thankful ALCOTT was born in 1748 in New Haven, New Haven, Conn. and died on 1 Mar 1839.x. Hannah ALCOTT was born in 1751 in New Haven, New Haven, Conn. and died on 1 Mar 1821.xi. Anna ALCOTT was born about 1753 in New Haven, New Haven, Conn. and died on 5 Feb 1822 in Wol cott, New Haven, Conn.xii. Stephen ALCOTT was born about 1757 in New Haven, New Haven, Conn. 18. Solomon CHATFIELD was born on 13 Aug 1708 and died in 1779. He married Hannah PIERSON on 12 Jun 1734. 19. Hannah PIERSON was born on 4 Aug 1715 and died on 15 Mar 1801. She is buried in Oxford Congregational Cemetery, Oxford, Conn. Solomon CHATFIELD and Hannah PIERSON had the following children: i. Joseph CHATFIELD was born on 4 Apr 1735 and died about 1795.9  ii.  Mary CHATFIELDiii. Hannah CHATFIELD was born about 1738.iv. Lois CHATFIELD was born about 1741.v. Eunice CHATFIELD was born on 6 Feb 1743 and died in 1823.vi. Rachel CHATFIELD was born about 1745 and died on 11 May 1778.vii. Comfort CHATFIELD was born about 1749.viii. Anna CHATFIELD was born about 1752 and died on 11 Sep 1853.ix. Comfort CHATFIELD was born about 1756 and died on 3 Nov 1798. 28. Joseph SEWELL was born on 15 Aug 1688 in Boston, Suffolk Co., Mass. and died on 27 Jun 1769 in Boston, Suffolk Co., Mass. He married Elizabeth WALLEY on 29 Oct 1713 in Boston, Suffolk Co., Mass. 29. Elizabeth WALLEY was born on 4 May 1693 in Boston, Suffolk Co., Mass. and died on 27 Oct 1713 in Boston, Suffolk Co., Mass. Joseph SEWELL and Elizabeth WALLEY had the following children: 14  i.  Samuel SEWELLii. Joseph SEWELL was born on 13 Jul 1719 in Boston, Suffolk Co., Mass. and died on 18 Aug 1719 in Boston, Suffolk Co., Mass. 30. Edmund QUINCY was born on 13 Jun 1703. He married Elizabeth WENDELL on 15 Apr 1725 in Boston, Suffolk Co., Mass. 31. Elizabeth WENDELL was born. Edmund QUINCY and Elizabeth WENDELL had the following children: 15  i.  Elizabeth QUINCY Sixth Generation (Great, Great, Great Grandparents) 32. John ALCOTT was born on 14 Jul 1675 in New Haven, New Haven, Conn. and died in Mar 1722 in New Haven, New Haven, Conn. He married Susanna HEATON on 8 May 1698 in New Haven, New Haven, Conn. 33. Susanna HEATON was born on 12 Apr 1680 in New Haven, New Haven, Conn. and died on 3 Mar 1736 in New Haven, New Haven, Conn. John ALCOTT and Susanna HEATON had the following children: i. Abigail ALCOTT was born in 1703 in New Haven, New Haven, Conn. and died in 1771.16  ii.  John ALCOCKiii. Elizabeth ALCOTT was born on 31 Jul 1708 in New Haven, New Haven, Conn. and died on 23 Jan 1782 in New Haven, New Haven, Conn.iv. Sarah ALCOTT was born on 11 Aug 1711 in New Haven, New Haven, Conn. and died in 1757.v. Stephen ALCOTT was born on 10 Aug 1714 in New Haven, New Haven, Conn. and died in Feb 1742.vi. Mary ALCOTT was born on 10 Aug 1717 in New Haven, New Haven, Conn. 34. John BLAKESLEE was born on 15 Jul 1676 in New Haven, New Haven, Conn. and died on 30 Apr 1742 in New Haven, New Haven, Conn. He married Lydia in 1696. 35. Lydia died on 12 Oct 1723 in New Haven, New Haven, Conn. John BLAKESLEE and Lydia had the following children: i. Elizabeth BLAKESLEE was born on 1 Mar 1702 in New Haven, New Haven, Conn.17  ii.  Deborah BLAKESLEEiii. Mary BLAKESLEE was born on 5 Apr 1720 and died about 1799. 36. John CHATFIELD was born on 8 Apr 1661 in Guilford, New Haven, Conn. and died on 7 Mar 1748. He married Anna HARGER on 5 Feb 1685 in Derby, New Haven, Conn. 37. Anna HARGER was born on 23 Feb 1668 in Stratford, Fairfield, Conn. and died in 1748. John CHATFIELD and Anna HARGER had the following children: i. Sarah CHATFIELD was born on 5 Dec 1686 and died on 20 Jun 1721.ii. Mary CHATFIELD was born on 23 Apr 1689.iii. Abigail CHATFIELD was born on 2 Sep 1693.iv. John CHATFIELD was born on 26 Feb 1697 and died on 30 Oct 1793.v. Samuel CHATFIELD was born on 28 Aug 1699 and died on 17 May 1785.vi. Ebenezer CHATFIELD was born on 4 Jul 1703 and died about 1789.18  vii.  Solomon CHATFIELD 38. Abraham PIERSON was born about 1680 and died on 12 May 1758. He married Sarah TOMLINSON. 39. Sarah TOMLINSON was born about 1690 and died on 12 May 1758. Abraham PIERSON and Sarah TOMLINSON had the following children: i. Sarah PIERSON was born on 19 Aug 1705 and died in 1750.ii. Abraham PIERSON was born on 28 Jul 1707 and died in 1781.iii. Mary PIERSON was born on 26 Oct 1712 and died in 1790.19  iv.  Hannah PIERSONv. Stephen PIERSON was born on 4 Mar 1720 and died in 1758.vi.  Barchua  PIERSON was born on 1 Dec 1726. Seventh Generation (Great, Great, Great, Great Grandparents) 64. Phillip ALCOTT was born in 1648 in Dedham, Norfolk, Mass. and died in 1715 in Wethersfield, Hartford, Conn. He married Elizabeth MITCHELL on 5 Dec 1672 in New Haven, New Haven, Conn. 6 5. Elizabeth MITCHELL was born on 6 Aug 1651 in New Haven, New Haven, Conn. Phillip ALCOTT and Elizabeth MITCHELL had the following children: 32  i.  John ALCOTTii. Thomas ALCOTT was born in 1677 in New Haven, New Haven, Conn. and died on 2 Apr 1757 in New Haven, New Haven, Conn.iii. Elizabeth ALCOTT was born on 6 Feb 1679 in New Haven, New Haven, Conn.iv. Phillip ALCOTT was born on 19 Nov 1681 in New Haven, New Haven, Conn.v. Agnes ALCOTT was born in 1683 in New Haven, New Haven, Conn. and died on 8 Feb 1782. 66. James HEATON was born about 1632 and died on 16 Oct 1712 in New Haven, New Haven, Conn. He married Sarah STREET on 20 Nov 1662. 67. Sarah STREET was born about 1640. James HEATON and Sarah STREET had the following children: i. Nathaniel HEATON was born on 19 Nov 1664 and died in 1725.ii. Abigail HEATON33  iii.  Susanna HEATONiv. Anna HEATON was born on 23 Dec 1682.